When Diarrhea Is More Serious Than You Think

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Those Who Invested In CTS Eventim KGaA (ETR:EVD) Five Years Ago Are Up 100%

Stock pickers are generally looking for stocks that will outperform the broader market. Buying under-rated businesses is one path to excess returns. For example, the CTS Eventim AG & Co. KGaA (ETR:EVD) share price is up 93% in the last 5 years, clearly besting the market return of around 1.4% (ignoring dividends). However, more recent returns haven't been as impressive as that, with the stock returning just 41% in the last year, including dividends.

So let's investigate and see if the longer term performance of the company has been in line with the underlying business' progress.

Check out our latest analysis for CTS Eventim KGaA

While markets are a powerful pricing mechanism, share prices reflect investor sentiment, not just underlying business performance. One flawed but reasonable way to assess how sentiment around a company has changed is to compare the earnings per share (EPS) with the share price.

During the last half decade, CTS Eventim KGaA became profitable. That's generally thought to be a genuine positive, so investors may expect to see an increasing share price.

You can see below how EPS has changed over time (discover the exact values by clicking on the image).

earnings-per-share-growth

It is of course excellent to see how CTS Eventim KGaA has grown profits over the years, but the future is more important for shareholders. You can see how its balance sheet has strengthened (or weakened) over time in this free interactive graphic.

What About Dividends?

It is important to consider the total shareholder return, as well as the share price return, for any given stock. The TSR is a return calculation that accounts for the value of cash dividends (assuming that any dividend received was reinvested) and the calculated value of any discounted capital raisings and spin-offs. Arguably, the TSR gives a more comprehensive picture of the return generated by a stock. In the case of CTS Eventim KGaA, it has a TSR of 100% for the last 5 years. That exceeds its share price return that we previously mentioned. The dividends paid by the company have thusly boosted the total shareholder return.

A Different Perspective

It's good to see that CTS Eventim KGaA has rewarded shareholders with a total shareholder return of 41% in the last twelve months. That's including the dividend. That gain is better than the annual TSR over five years, which is 15%. Therefore it seems like sentiment around the company has been positive lately. In the best case scenario, this may hint at some real business momentum, implying that now could be a great time to delve deeper. It's always interesting to track share price performance over the longer term. But to understand CTS Eventim KGaA better, we need to consider many other factors. To that end, you should be aware of the 1 warning sign we've spotted with CTS Eventim KGaA .

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But note: CTS Eventim KGaA may not be the best stock to buy. So take a peek at this free list of interesting companies with past earnings growth (and further growth forecast).

Please note, the market returns quoted in this article reflect the market weighted average returns of stocks that currently trade on German exchanges.

Have feedback on this article? Concerned about the content? Get in touch with us directly. Alternatively, email editorial-team (at) simplywallst.Com.

This article by Simply Wall St is general in nature. We provide commentary based on historical data and analyst forecasts only using an unbiased methodology and our articles are not intended to be financial advice. It does not constitute a recommendation to buy or sell any stock, and does not take account of your objectives, or your financial situation. We aim to bring you long-term focused analysis driven by fundamental data. Note that our analysis may not factor in the latest price-sensitive company announcements or qualitative material. Simply Wall St has no position in any stocks mentioned.

Have feedback on this article? Concerned about the content? Get in touch with us directly. Alternatively, email editorial-team@simplywallst.Com

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Ebola Can Hide In Humans For Years Before Killing Again, New Research Finds

New genetic research appears to confirm a fear scientists had about a deadly outbreak of Ebola in Guinea this February: The source of the outbreak was likely the dormant remnants of the virus in a survivor who caught it at least five years earlier. The discovery could complicate efforts to contain the viral emerging disease.

In late January 2021, a 51-year-old female nurse in the West African country came down with Ebola (formally known as Ebola virus disease, or EVD). Like many victims, she initially experienced vague symptoms like headache, nausea, and general weakness. Though she was hospitalized, her doctors unfortunately misdiagnosed her with malaria and a salmonella infection, and she was sent home after two days. She became sick again at home and died three days later.

Following her death, her husband and other family members who attended her funeral became sick as well, and four would ultimately die. These cases finally alerted national health officials. By mid-February, blood samples confirmed the outbreak, and health care workers rushed to control it. Between February to June 2021, when the outbreak was formally declared over, 16 confirmed cases were reported, along with 12 deaths.

Typically, Ebola outbreaks start with zoonotic transmission from an infected animal to a person, with certain bats thought to be the primary host of the virus. But early analysis of the first blood samples collected from victims during this outbreak suggested something else was going on. The virus seen in their blood looked very similar to a variant collected from survivors of the 2013-2016 West African outbreak of Ebola, the largest and most deadly epidemic of the virus to date, with over 11,000 deaths. Scientists soon suspected the virus had somehow reemerged from a human host years later and was killing again.

This new study, published Wednesday in Nature, all but seals that hunch as being right. Researchers in Guinea, France, and Germany conducted next-generation genetic sequencing of the virus collected from 12 victims of the outbreak, using it to construct complete or near-complete genomes of the virus. They then used this information to build a family tree of the strains and compared them to the virus that was floating around five years ago.

The virus found in Guinea this year was indeed closely related to the virus seen during the earlier West African outbreak, the authors said, indicating "that the new outbreak was not the result of a new spillover event from an animal reservoir." Moreover, they didn't find signs of much genetic divergence between then and now, suggesting that it wasn't being heavily transmitted between humans all that time. Instead, the source virus may have been causing a persistent but slow-moving infection with reduced reproduction in a survivor, or it could have gone completely latent, then reactivated for some unknown reason, making the survivor contagious again.

Scientists have known since the West African outbreak that the virus can survive unnoticed in victims for some time after they've fully recovered, especially in pockets of the body where the immune system is less active, such as the eyes or sperm. But the longest known time between someone contracting the virus and passing it on to someone else or becoming sick again, prior to this, was around a year and a half. A much larger window of transmission for the virus means that communities may be more vulnerable to future outbreaks than currently thought. It may also make the lives of survivors, who already tend to experience discrimination and prejudice from others, even harder, the authors warn.

"The human origin of the 2021 EVD outbreak, and the associated shift in our perception of EBOV emergence, call for careful attention to survivors of the disease," they wrote. "The concern that survivors will be stigmatized as a source of danger should be a matter of scrupulous attention."

In this case, it's possible that the unknown patient zero may have transmitted the virus during sexual intercourse. But given that the first new victim was a nurse, the patient may also have experienced a reemergence of symptoms that led to a health care visit and transmission at that point. The nurse herself may have had a dormant infection return, though that's less likely, since she had no previously documented case.

On the big-picture level, the authors say these findings should fuel research into finding a way to keep survivors virus-free and protecting communities after an outbreak has subsided.

"In addition to the importance of appropriate healthcare measures focused on survivors, the late resurgence of the virus also highlights the urgent need for further research into potent antiviral agents that can eradicate the latent virus reservoir in patients with EVD, and into efficient vaccines that provide long-term protection," they wrote.

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Aaron Chalmers' Ex Talia Gives Heartbreaking Update On Seriously Ill Son's Surgery

AARON Chalmers' ex Talia Oatway has shared a heartbreaking health update on their one-year-old son Oakley after five months in hospital.

Talia revealed earlier this week that her little boy had to go back into surgery after a hole was found in his skull, causing brain fluid to leak out.

Aaron Chalmers' ex Talia explained why her son had been in hospital for over a monthCredit: Instagram/talia.Oatway

The brave one-year-old is receiving treatment after having four surgeriesCredit: Instagram/talia.Oatway

The influencer and model explained that Oakley has been receiving treatment in hospital for over a month now.

Talia listed the medical reasons why her son had been admitted for so long.

Alongside a sweet clip of her son in bed, the mum-of-three wrote: "My champ. Lots of messages regarding why we are here still.

"Oakley had the FOA on the skull on the 13th May.

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"Unfortunately with Apert Syndrome, the operations don't always go to plan.

"Oakley had to have this surgery to help his brain, development and to protect the eyes.

"This wasn't done out of choice."

Watch as emotional Talia Oatway gives health update on son with Aaron Chalmers

Talia concluded: "Everyday he shows me what true strength is."

Just yesterday, Talia admitted that she had been "struggling" and "crying a lot" after a tough week.

On Saturday, Talia told her followers that Oakley had to have surgery again to find out why his cerebrospinal fluid (CSF) was leaking.

She wrote on her Instagram stories: "Putting your child into theatre never gets easier. They are going to try and see where the leak of CSF is coming from. They won't put a shunt in yet as Oakley is still on strong IV antibiotics.

"There will be a meeting next week to discuss long-term plans if this carries on leaking. They mentioned he may have another drain fitted today."

She then updated fans today, explaining that Oakley was out of surgery and the cause of the leak had been found.

Talia wrote: "Another surgery last night. They found a hole under Oakly's skull which was leaking CSF. He's got a lumbar drain in for now to help drain the CSF.

"There will be a meeting on Monday to discuss the next step which will probably lead to another operation which could be a shunt."

Later in the day, she shared another picture with her pride and joy from his hospital room, calling him "the strongest little boy".

Oakley suffers from a rare genetic condition called Apert Syndrome, which affects how bone cells grow and divide.

Recently, Oakley was rushed into hospital with life-threatening sepsis, which occurs when the body's reaction to an infection affects the tissues and organs.

He underwent surgery at the time to drain the infection from his head.

Talia said at the time that she was "living a nightmare" and just wanted Oakley "safe and home."

"I'm not going to lie this week has been the worst week of my life. To watch my son be intubated, 2 surgeries in a matter of days, fighting sepsis, drain in his back, leaking fluid from his head, fluid resuscitation, blood transfusions, I am struggling here.

"Oakley has a long road ahead, this is a lifelong journey and it isn't going to be easy. But my boy, you have shown me how strong you are!

"You are brave and determined and I love you more than you'll ever know. I hope that by sharing our journey I can raise awareness and support other medical families."

What is Apert syndrome?

APERT syndrome, also known as acrocephalosyndactyly, is a rare disorder that is named after the doctor who first discovered it in the early 20th century.

It is a genetic condition and is caused by a mutation of the FGFR2 gene.

This affects how cells in the body - namely bone cells - grow, divide and die.

Children born with Apert syndrome have a characteristic appearance, which is caused by the bones in the skull and face fusing and not growing in proportion, according to Great Ormond Street Hospital.

It can increase a child's risk of hydrocephalus, which results in pressure building on the brain, and it can also cause Chiari malformation, where the base of the brain is squeezed.

Other complications include breathing difficulties and heart problems, which require life-long monitoring.

The condition is said to occurs in one in every 65,000 to 88,000 births and a child's outlook can vary greatly depending on the severity of symptoms

Oakley suffers from a rare genetic condition called Apert SyndromeCredit: instagram/@aaroncgshore

Ebola Virus Disease